Glaucoma and Ocular Hypertension

Glaucoma and Ocular Hypertension

Ocular hypertension is intraocular pressure [the fluid pressure of the aqueous humor inside the eye] that is higher than normal in the absence of optic nerve damage or visual field loss. Normal intraocular pressure is normally defined as being between 10 mmHg and 21 mmHg. When the pressure is elevated there is a considerable risk factor of developing glaucoma.

Ocular hypertension are mostly treated with acetazolamide pilocarpine and timolol. There are also other, less commonly used, but perhaps more pleasing alternatives, such as medicinal cannabis.

Glaucoma is considered as an eye disorder in which the optic nerve has suffered damage, which bas a result causes permanent vision problems with the eye/s. It can end up progressing to complete blindness if not treated effectively. While Glaucoma is associated with the increase of pressure in the fluid of the eye quite often, it is not the only potential cause of the disorder.

It should also be remembered that not everyone who has high levels of pressure in their eyes will suffer from nerve damage. Many people can have high pressure for years with no damage being caused. While other with relatively low pressure can still end up with nerve damage being caused as a result.

Glaucoma is often categorized into two main types – “Open Angle” and “Closed Angle”. Open angle, chronic glaucoma usually progresses at a slow rate and there may be no loss of vision until the disease has progressed significantly. While closed angle glaucoma can appear suddenly and can also be accompanied with quite a high level of pain. The loss of vision can also progress rapidly. Fortunately, the discomfort associated with this type of glaucoma usually results in people seeking medical attention quickly, resulting in an increased opportunity to prevent permanent damage.


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Posted by Dr. John on Jan 22 2011. Filed under Breaking, Health. You can follow any responses to this entry through the RSS 2.0. You can leave a response or trackback to this entry

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